Skip to main content
Fig. 1 | BMC Pharmacology and Toxicology

Fig. 1

From: Role of uranium toxicity and uranium-induced oxidative stress in advancing kidney injury and endothelial inflammation in rats

Fig. 1

Changes in the oxidative stress parameters and serum and tissue uranium content in rats of different groups. Notes The circular dot represents a mean of this parameter in the corresponding group. The mean levels of Tissue U and Serum U in the NS group were excluded from the Jonckheere–Terpstra test because they did not represent the initial uranium concentration after uranium exposure. After uranium exposure, the levels of TOS and OSI show an upward trend, whereas the levels of TAS and Tissue U show a downward trend; the content of Serum U was raised at 72 h. After one-time exposure to uranium, kidney excretion and metabolism could gradually remove uranium from the body, and kidney absorption and metabolism might also participate in the excretion of uranium deposited in the kidney tissue

Back to article page